Diabetes is considered a syndrome rather than a disease by modern medicine. It remains a companion to the grave - once diabetic always diabetic is the order of the day. Due to different reasons the Beta cells of Pancreas get impaired and adversely affects the quality or quantity or both of Insulin being produced in the body - partially in case of Type 2 & totally in case of Type 1 Diabetes. Due to the insufficiency or inefficiency of insulin produced by pancreas, blood sugar levels go up degrading strength & health of human body by starving all cells. Damaging vulnerable organs like Kidney & Heart makes diabetes fatal; whereas impairment of Retina of eye, cerebral haemorrhage/stroke, gangrene of foot/lower-leg etc make rest of life miserable.
Diabetes is seen to be progressive, degenerative & incurable. This is because the cause (impairment of pancreas) is never treated; only its effect (blood sugar increase) is looked into and taken care of.
The author hence left the beaten track when he was diagnosed diabetic & took a drastically different approach instead, based on the enlightenment imparted by an Aayurveeda exponent and his own exposure. He designed a diet based on Long Wheat Mash as the staple part & a life style appropriate to a diabetic. Within weeks of the special regimen, FBS & PPBS came down steadily & settled at normal values. But it took still longer for the HbA1c to descend to normal range. Then he confirmed cure by OGTT, and reconfirmed it with a repeat OGTT at another NABL, ISO & ACHSI certified lab. He further assured the cure by a serum insulin test also.
In order to ensure that the illness or syndrome has not returned by switching back to normal diet, he gets FBS & PPBS tested every month and HbA1c quarterly from 2009 onwards.
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Breaking news[edit | edit source]
Is this an attempt to snatch the creatorship of Long Wheat from MOTHER NATURE?
Efficacy of Long Wheat (LW) in Diabetes Control[edit | edit source]
Scientific evidence corroborating Georgedappilly's claim (2009) on hypoglycemic efficacy ofLong Wheat had been lying dormant for one and a half decades in a Scientific Paper [2]. Unfortunately the conclusion of the paper was in the negative on hypoglycemic effect of LW. This turned out to be a blessing in disguise for Georgedappilly. A staunch opponent of Long Wheat Mash Diet Regimen LWMDR dug out the 14 year old study paper. I followed the link & at the very first glance itself noticed the positive fact contained in the included data (TAble II of the paper) [2]. This is a panacea for Diabetics but a dust in the eye for some others!
The point that authors left unnoticed in the paper & caught my attention as very important is: the increase in blood sugar that happened due to the experimental diet in two fortnights was reverted by the next one fortnight in the case of Group I (who used Dicoccum whole Wheat flour) but not so in the case of Group II (who used Bread Wheat flour)!!!
This study also indicates that even LW flour in the form of unleavened bread works (not only mash!) in regulating the Blood Sugar of Diabetes patients.
The referred paper is certainly a great encouragement to all those who are on LWMDR as well as those who are contemplating on trying LWMDR.
Usual life-cycle of the dreaded disease named Diabetes as understood by me
Most persons think & propagate that Diabetes is a syndrome and not a disease; hence incurable. I totally disagree with both. Diabetes is also considered to be caused by the impairment of Pancreas (to be more precise, of Beta-cells which produce the hormone Insulin). From the time of reading about Insulin resistance (IR) I beg to differ with the Pancreas-BetaCell theory also.
Insulin Resistance, one of the triggers and sustainers of Diabetes[edit | edit source]
Insulin is a hormone produced by the beta cells of gland Pancreas. Initially it is attached to C-peptide as "Siamese twins" and gets detached on maturing in blood. Half life period of endogenous Insulin is estimated to be 5 minutes (whereas that of C-peptide is 30 min). It is Insulin that helps Glucose molecules enter cells from Blood easily and normslise the Blood Glucose level.
When the quantum of Glucose is more than what is needed to meet the immediate energy requirements, the excess quantity is stored as Glycogen in Liver and further as fat at other parts of the body.
When the storage spaces overflow, cells find it difficult to receive Glucose at the prevailing rate and signal the Insulin receptors to reduce their sensitivity. This is known as Insulin resistance (IR).
IR in turn reduces Glucose (G) absorption/removal from Blood (B). In my opinion this phenomenon is Diabetes. This leads to increased BG levels and a host of subsequent problems generally known as Diabetic complications.
Misinterpreting the increased levels of BG (alias Blood Sugar - BS) being caused by shortage of Insulin, body intelligence signals Pancreas to enhance production. This may give a temporary relief but leads to higher levels of Insulin & Glucose in Blood as the IR still prevails.
Increased BS levels lead to further increase in IR! Aaaaand the vicious circle continue obviously till......
Misunderstanding the increased levels of BG/BS being caused by shortage of Insulin, patients are advised and resorts to:
A. physical exercises,
B. oral therapies to whip Pancreas for producing more Insulin,
C. inject extraneous Insulin,
etcetera in order to force cells to absorb more Glucose from Blood.
Being unaware of IR and due to taste and enjoyment considerations, patients continue to take sweet/starch in improper quantities. This in turn cause further hardships to cells. This leads to further increase of IR to face the enhanced challenge. This further worsens the situation and pushes the BS level to still higher values! Subsequently we enhance the therapy level. This vicious circle continues till the "unstable phase" alias "Brittle Diabetes" emerge!!
Four cases compared to show role of IR in trigger & sustenance of DM
Parameter | Case1 Me
Diab cured |
Case2 IFG Relative | Case3 Wife of an acquaintant | Case4 Friend on Insulin inj | Case5 Friend's sonSurApn on exercise only | Case6 Boban Relative | Remarks |
---|---|---|---|---|---|---|---|
Date | 03Aug2016 | 26Sep2016 | 11Nov2016 | ..Dec2014 | 30Jan2021 | FebMar2022 | |
FBS* | 95 | 109 | 218 | 150 | 114 | 127 | * mg/dL |
PPBS mg/dL | 86 | 79 | 190.7** | 200* | 116 | 170(ND/Calc) | * Average **eAG (A1C) |
HbA1c % | 5.0 | ND | 8.1 | 9+ | 6.30 | 6.7 | ND=Not Done |
FSI micro U/mL | 5.41 | 11.2 | 15.6 | 200+ | 40.12 | 6.83 | Range 2.6 - 24.9 |
TriG mg/dL | 54 | 156 | 91.4 | 143 | 169 | 131 | <150 |
HDL mg/dL | 59 | 63 | 75 | 49 | 32 | 42 | > 40 |
IR(1) | 1.269 | 3.014 | 8.39 | 74 | 11.29 | FSI×FBS÷405 <=1.0 | |
IR(2) | 0.915 | 2.476 | 1.218 | 2.918 | 5.28 | TriG÷HDL range <=2.0 | |
Insulin Inj | Nil | Nil | ? | 30-0-30 | Nil | Nil | |
Tab1 | Nil | Nil | ? | Glucobay (50) 1-1-1 | Nil | Nil | |
Tab2 | Nil | Nil | ? | Galvus met (50/1000)☆ 1-0-1 | Nil | Nil | ☆>Vildagliptin 50 + Metformin 1000 |
Tab3 | Nil | Nil | ? | Nil | Nil | Nil |
Analysis[edit | edit source]
My FBS is less than 100 with fasting Serum Insulin (FSI) only 5.41. My relative has FBS more than 100 even with FSI 11.2; more than double of mine. This is a conspicuous exhibition of IR & Insulin being more in Blood even at beginning of Diabetes! She is only at impaired fasting Glucose (IFG) stage, not full blown diabetic according to modern medicine standards!
Wife of my acqaintant has FBS 218, more than both the cases mentioned above even with FSI 15.6; which is almost 300% of mine and 150% of the second case. This again exhibit the role of IR on Diabetes & even excess Insulin being unable to hold Blood Sugar at normal levels! She is on Diabetic medication as well!
My friend had FSI 200+ with three Insulin shots daily; third dose decided based on Supper PPBS! In spite of all the "exercises" he has FBS around 150!!
Fasting Blood Sugar (FBS) of case1 is the least of the four, in spite of the fasting serum Insulin (FSI) being the lowest!!!
Comments are welcome.
Effect of Diabetes on subsystems &/or components of human body[edit | edit source]
PRIMARY: (usually go unnoticed)
- Blood sugar level exceeding upper limits (FBS >100 mg/dl, PPBS>140 mg/dl)
- HbA1C increasing (normal range: 4.2% - 5.7%)
- Cells being deprived of Oxygen & Glucose; getting filled with Carbon dioxide. (Glucose deprivation by Insulin Resistance and others by Glycosylated Haemoglobin alias HbA1c)
SECONDARY: (obvious & leads to detection)
- Frequent urination, persisting &/or unclenched thirst & repeating hunger.
- Quick & unexplainable weight loss.
- Persistent tire & fatigue unresolved even by food intakes and rest.
- Possible misunderstanding among young couples sometimes leading to family breakdown due to diabetic sexual dysfunction
TERTIARY: (affecting quality of life & confidence)
- Numbness in lower parts of legs (ankles & feet).
- Even minor skin injuries developing to prolonged wounds.
- Loss of sensation. (Even ant-bites on wounds being not sensed.)
- Liver enlargement & associated/subsequent problems
FIERCE FINALE: Diabetic neuropathy sometimes culminating in:-
- Diabetic neuropathy gangrene of legs – leading to amputations.
- Diabetic retinopathy – irreversible loss of sight.
- Diabetic nephropathy – irreparable kidney damage.
- Diabetic brain hemorrhage (Stroke)– lifelong total or partial paralysis.
- Atherosclerotic Heart Disease (Silent Angina) – the FINAL event.
The beaten track of Diabetes management[edit | edit source]
Stage 1: Starts with exercise & diet control (quantity restriction & Glycemic Index based food swapping).
Stage 2: Progress to low dose medication usually ½ Tab once daily in addition to stage 1 practices.
Stage 3: Advance to higher doses of Tabs + Stage 1 practices.
Stage 4: Switch to stronger Tabs in Stage 3 practices.
Stage 5: Progress further to INSULIN injection (initially once daily, then twice, thrice & finally with every meal) in addition to Stage 1 practices.
Stage 6: Then combination of a few of the above.
Stage 7: Combination (union) of all the above (diet control, exercise, tabs & insulin injections).
Stage 8: Penultimate, the UNSTABLE phase where prescribed dose of medicines pulls to hypoglycemia & reduced dose shoots the blood sugar up! (this topples the peace of mind as well as courage). This phase is known as Brittle Diabetes among medical professionals.
Stage 9: Finally the BREAKDOWN phase when the patient gives up all restrictions on diet and lifestyle in utter despair, realising the futility of decades long struggling and compromises.
- There are two more stages which cannot be uniquely placed in the above sequence. These are relatively recent developments.
Bariatric surgery. This surgical procedure reduces the food intake capacity and gives satiety with lesser quantity. That in turn reduces Blood Sugar lelevel as well as body mass.
Insulin Pump Implantation. As indicated by the name a pump that sends Insulin to bloodstream is implanted on the patient. It works on battery power and has a refillable/replaceable storage of Insulin. Pumping rate is presetable. The approximate size is comparable to a matchbox.
Some Diabetic Neuropathy Gangrene cases[edit | edit source]
Anxious waiting! Wondering what surprise this time? Fingers or foot!?
=
A MalayalaM news paper report
Met two Diabetic patients suffering severe pain due to gangrened foot, on a railway platform. This sight forced me to a news paper office near by. I explained the encounter & my different experience with LWMDR.
The reporter is nice & was patient enough to listen to me for 2 hours!
They published reports at their printed & online editions. A Malayalam news paper report.
A different experience - Diabetes cured
The diabetes mellitus of User:Georgedappilly is cured.
Sample lab results are given there. Details (in vernacular language MalayalaM) are placed there. The English versionof procedures, experiences & results is released.
Type 2 diabetese is supposed and popularly accepted by experts world over to be incurable. That one is what is cured in him by diet management (appropriate technology).
Comment: To monitor your condition, preferably have periodical test using a suitable test kit and/or consultation to doctors.
My latest Blood test report
This is done as part of routine quarterly cardiology check up. Completing a decade of diagnosing Diabetes. All the tested parameters are normal, in range.
Sl No. | Parameter | Test method | Result/value | Unit | Ref. Range |
---|---|---|---|---|---|
1 | FBS | Hexokinase | 95 | mg/dL | 70-100 |
2 | Cholesterol | CHOD PAP | 129 | mg/dL | <200 |
3 | Triglycerides | GPO Enzymatic | 54 | mg/dL | <150 |
4 | HDL Cholesterol | Homogenous enzymatic | 59 | mg/dL | >40 |
5 | LDL Colesterol | Homogeneous enzymatic | 62 | mg/dL | <100 |
6 | VLDL | Calculated | 10 | mg/dL | 10-30 |
7 | Cholesterol/HDL ratio | Calculated | 2 | ||
<4.5 | |||||
8 | Blood Urea Nitrogen | GLDH Urease kinetic method | 9.3 | mg/dL | 8-23 |
9 | Creatinine | Jaffes Kinetic method | 1 | mg/dL | 0.60-1.30 |
10 | Total Leukocyte Count | TC | 6300 | cells/cumm | 4000-10000 |
11 | Platelet Count | ||||
156 | thousand/cu.mm | 150-400 | |||
12 | Haemoglobin% | Hb | 13.5 | g/dL | 13-16 |
13 | Packed Cell Volume | PCV | 42.9 | % | 40-54 |
14 | Glucose 2 Hr PPBS | Hexokinase method | 86 | mg/dL | <140 |
15 | Insulin fasting | ECLIA | 5.41 | micro U/ml | 2.60-24.90 |
A very brief description of George's experience[edit | edit source]
A blood test on Jul 07, 2006 showed FBS 118 mg/dl & PPBS 197 mg/dl. Doctor declared him diabetic and prescribed Amaryl 1.0 mg 1/2-0-0. With doctor's permission he went on diet management without taking medicine. Regular periodic check ups showed positive changes.
On Jan 24, 2009 switched from FBS-PPBS monitoring to FBS, PPBS & HbA1C (HPLC in D10 Biorad)monitoring. That day HbA1C showed 6.3% (reference range: 4.5%-6.3%). Next on Apr 22, 2009 it came down to 6.1% and on Jul 25, 2009 to 5.9%!!!
Then he decided to go for a GTT and had it on Sep 10, 2009. From FBS 85 mg%, with an oral glucose load of 75 gms it went to 155 at 1/2 hr, to 160 at 1 hr, came down to 135 at 1.5 hrs, to 100 at 2 hrs and to 90 at 2.5 hrs!!!
According to endocrineweb (http://www.endocrineweb.com/diabetes/diagnosis.html) and Wikipedia these results show that his diabetes is cured.
In order to make things doubly sure, he took a second GTT at another lab (of an N A B H, I S O & A C H S I accredited hospital). That corroborated the previous test result.
HbA1C & Serum Insulin
Date | FBS(mg/dl) | PPBS(mg/dl) | HbA1c(%) | Remarks |
---|---|---|---|---|
07.07.2006 | 118 | 197 | Not tested | Diagnoded Diabetes |
20.01.2009 | 108 ## | 161 ## | 6.3* | *24.01.2009 |
22.04.2009 | 115 ## | 74 ## | 6.1 | |
......Till this | Diabetic | .......After this | NOT Diabetic | Hence SPECIAL regimen DISCONTINUED |
09.07.2009 | .. | 100# | .. | .. > Not tested; # > 6 Sweets |
25.07.2009 | 108 ## | 74 ## | 5.9 | |
10.09.2009 | 85 | 100$ | .. | $ GTT-1 |
14.09.2009 | 109 | 118$$ | .. | $$ GTT-2 |
07.10.2009 | 97 ## | 98 ## | 6.1 | |
27.01.2010 | 100 ## | 105 ## | 6.0 | |
28.04.2010 | 105 ## | 131 ## | 6.2 | |
31.07.2010 | 107 ## | 88 ## | 6.2 | |
07.12.2010 | 96 ## | 94 ## | 5.9 | ## Hexokinase method |
25.03.2011 | 97 | 145 | 6.1 | Serum Insulin (Eclia) 10.03 micro U/ml. ---- Ref. Range: 2.6 - 24.9 |
12.07.2011 | 104 ## | 99 ## | 6.2 | .. |
14.10.2011 | 89 | 92 | 6.3 | .. |
13.01.2012 | 100 ## | 85 ## | 5.9 | Children vacation, X'mas, Newyear |
09.11.2012 | 104## | 113## | 5.9 | Test at ACHSI and NABL certified lab |
22.02.2013 | 100 ## | 104 ## | 5.9 | PPBS after BF of {3 Iddali (alias Idli) = 300 k cal + 1 cup daal saambaar + 0.5 cup coconut chutney + coffee with milk & sugar} |
<17.04.2013 | 92 | .. | 5.9 | |
07.08.2013 | 99 ## | .. | 5.6 | Routine quarterly check up |
06.11.2013 | 103 ## | .. | 5.9 | .. |
22.11.2013 | 93 GM (1)
94 GM (2) |
.. | .. | (1) 9Hr 45 min FASTING
(2) 14 HR FASTING |
31.11.2013 | .. | 127 GM | .. | |
26.08.2017 | 94 | .. | 5.8 | Routine cardiology quarterly checkup |
.. | .. | |||
.. | .. | |||
.. | .. | |||
.. | .. |
Long Wheat Mash Diet Regimen (LWMDR)[edit | edit source]
... #LWMDR... A name (GEM diet) was coined during March 2013 by a gentleman who is diabetic for more than two decades - 1991 onwards. He was administering 99 units of insulin & 1700 mg Metformin tab daily. He is very happy with the relief achieved by following my special diet. The word he chose to denote his experience, by following the diet detailed below, right from week one is amazing. It was then that he coined that name. The improvement continue(s/d) through the 11th week (June first week of 2013) also seamlessly.
But a Google search revealed that there is another diet existing with the same name, for weight reduction. Hence the need of an unique name being imperative, this new name - Long Wheat Mash Diet Regimen (LWMDR) - is chosen to avoid duplication/confusion.
Modus operandi[edit | edit source]
- In order to obtain initial conditions, get FBS, PPBS, HbA1C, Fasting Serum Insulin, Lipid profile, Serum Creatinine, E S R & C-peptide tests done before hand.
- Facilitate required (optimum-NOT maximum) rest to pancreas by TOTALLY AVOIDING sweet intake; avoid fruits as well till getting CURED. Avoid Maida flour, softened with Alloxan as it destroys Beta cells of Pancreas.
- Substitute break fast and dinner by long-wheat kanji (porridge/mash) with a pinch of salt as required (or Chapati, Puttu, unleavened bread etc), curries (hot/sour side dishes - with no sweet, no starchy vegetables). Continue lunch as it is (with restrictions on oil, fat, starchy vegetables like potato, tapioca etc & sweet). Take all the three meals stomach full. As body extracts micro medicine from LWM, intake is to be equispaced & voluminous; hence LWM swapped with staple part of breakfast & dinner.
- Reduce food intake frequency by postponing &/or combining and filling the gaps with plain water, buttermilk, fresh lime juice without sugar, vegetable salads etc. Most of the root-vegetables are rich in starch. May eat or drink more of LWM if number of intakes is three (3) or less including refreshments (snacks, tea, coffee, cool drinks etc.) in a day. Reduce oil & fat intake.
- Brisk, medium or at least normal walk preferably for an hour daily.
Caution
- Buy & use only unbroken long-wheat lest you should be cheated.
- If blood sugar does not start decreasing in a month, avoid smoking & liquor if you are in the habit of having those.
- Monitor blood sugar regularly - PPBS weekly, FBS at least monthly & HbA1C quarterly.
- If already on tab. or insulin inj. one must continue those & monitor blood sugar more frequently, preferably daily, at least for a week. Else one may slip into low blood sugar problem (hypoglycaemia?). As the FBS approach (not reach) lower limit (70 mg/dL or 3.89 mmol/L), reduce medication in small quantity either in consultation with your medical adviser or based on your judgement in conultation with me. Continue tapering down medicines till doses reach zero. Continue LWMDR till confirming cure by FBS, PPBS, HbA1c, OGTT, Serum Insulin & C-peptide tests after being ON NORMAL DIET for a few weeks.
- Neglecting any of the above eight points (four of modus operandi and four of caution) is similar to removing one of the legs of a table.
As you approach CURE, progress will be slower.
- In the case of advanced & prolonged diabetics having large medication, improvement (blood sugar reduction) will be visible on a daily basis.
- In moderate cases progress will be shown on a weekly or monthly basis only.
- Initial cases or those who have returned to near-normal values will see only monthly or quarterly betterment.
- Once blood sugar is normal, the cure process starts. Its progress will be still slower because the damaged organ (pancreas) is to get repaired.
- First improvement will be in PPBS, then comes FBS & subsequently HbA1c.
Why use only whole-grain Long Wheat?
- Broken or floured LW can be undetectably adulterated. It will be ineffective, in curing Diabetes, proportionate to the extent of adulteration.
- Adulteration is usually done using cheaper alternates for financial gain of manufacturer/trader & loss to buyer/consumer.
- Over & above these, broken/floured & packaged LW is almost twice as costly as wholegrain, in many places.
- Most important: Static charge accumulation by dry grinding may damage the curative ability of LW. If the static charge from the hand of a tradesman or engineer can damage man-made VLSIs & computer chips, the possibility of LW getting damaged can not be ruled out.
Most of the statements in this whole article (not this section alone) are my personal experiences, observations & inferences. Those are subjected only to my logical thinking and not to the so called rigorous scientific study or analysis.
Some remarks
- Diabetes (Type II) is considered to be mainly inherited from members up the family tree. Obesity, side effect of some medicines, sedentary life style, physiological or psychological shocks and some unknown reasons are also regarded as its causes.
- The victims as well as phisicians feel, belive & tell it can only be (supposedly) watched, monitored & controlled carefully,
- and have to teach & learn to live with it for ever.
- But this User's experience indicate that diabetes is more caused than happening.
- and can be prevented/cured and need not live with it for ever.
- The opinion that any amount of sweet or starch intake is OK until one become diabetic is NOT right.
- Pancreas is not meant to work 24X7 like the heart & lungs. It needs rest! Frequent food intakes strains it, instead.
However meticulous be the maintenance and good be the control of one's Diabetes, its degenerative feature DO persist and damage vital organs (like Kidney, Eyes, Heart,...) in due course.
That is why a diabetic patient passes away every 10 seconds (http://www.qda.org.qa/output/page4.asp). It is NOT SO after cure.
Duration required for diabetes cure[edit | edit source]
Presently (Nov 2009) data available is with regard to a single case of the User:Georgedappilly. It took three (3) years (from July 2006 to July 2009) to get his diabetes cured completly. His being the maiden case, many pit-falls occurred in the process. In future, the duration can be brought down because many mistakes are known now and these can be avoided in coming cases.
The pit-falls[edit | edit source]
Mistaking normal FBS & PPBS values for CURE
- Blood sugar history from pre-diabetic to post-diabetic phase (FBS blue & PPBS red)
- On completion of eight(8) weeks of special regimen, as the PPBS and FBS came below 140mg% and 105mg% respectively, he marginally relaxed the food restrictions. This moved PPBS above 140mg% in just twelve (12) days (!) {where as it took more than forty (40) days for the FBS to cross the 105mg% mark}.
- PPBS fell even below FBS level (105mg%) by Jan 2007 (by reintroducing diet control) and remained there till Feb 2007. This was misunderstood as CURE and again relaxed the intake constraints. By a lapse of four (4) months, even though FBS remained much below 105mg% (at June beginning) PPBS shot up above 140 mg%.
- On reimposing the restrictions, once again PPBS dropped down even below 105mg% (FBS level) and continued there for three months (12 weeks!). This again was wrongly interpreted as a cure and led to giving up of restrictions. This pushed up the PPBS to 192mg% by July 2008 (even though FBS continued near 105mg%).
- From mid August to end December 2008, PPBS remained below 140mg% (PPBS upper limit) & FBS continued near or below 105mg% (FBS upper limit). For a fourth time he was misled by these low FBS-PPBS values. As it should have happened, he relaxed diet control and ended up in PPBS reaching 161mg%.
- Reintroducing of diet control this time (Jan 20, 2009 onwards) really did the job. After five (5) months he got an opportunity to subject his pancreas to a qualification test.
My experience with FRUITS, during diabetes
Plantain fruit
- On Jul 07, 2006 my FBS & PPBS were 118 & 197 respectively. My cardiologist prescribed Amaryl. With his permission I started a new regimen of modified diet (which later on others christened as GEM Diet). In three weeks (on Aug 01, 2006) those values moved to 120 & 155. In another month (on Sep 01, 2006) those came down to 100 & 135; both in range or NORMAL.
- Mistaking this as cure re-introduced sugar to morning & evening coffee and restarted taking one plantain fruit after dinner. In eleven days (on Sep 12, 2006) PPBS rose to 155.
- As two sweet items (sugar & fruit) were introduced simultaneously, pinpointing the culprit was impossible. Plantain fruit being natural unprocessed sweet & presuming that sugar is the culprit, withdrew sugar & continued the fruit intake. In another three weeks (on Oct 04, 2006) FBS & PPBS registered 102 & 160 respectively.
- Even though both values are higher than their previous ones, the pictorial representation shows that the gradient (rate of growth) is lesser now. Even though this clearly indicated that both sugar and fruit increases blood sugar, I decided to continue taking plantain & avoid sugar for one more month. This was to provide time for the body to stabilise things and hoping that natural/fruit sweet may be harmless for diabetics as ALL 'EXPERTS' say. Next month's test results PROVED THOSE EXPERTS WRONG!! On Nov 07, 2006 FBS & PPBS were 121 & 166 respectively.
- Obviously there was no option other than to discontinue fruit intake also. In one month (on Dec 07, 2006) FBS & PPBS were 91 & 115 mg/dl. In another month (on Jan 02, 2007) those dropped to 80 & 98 and in one more month (on Feb 03, 2007) even though FBS marginally fluctuated up (to 83) PPBS decreased to 96 mg/dl.
- As both FBS & PPBS remained VERY LOW for two months, again reintroduced sugar in coffee & one plantain fruit at dinner. After two months (on May 29, 2007) while FBS maintained low normal value of 85 PPBS marked an increase to 155.
- Again withdrawing sugar & fruit brought back PPBS to 100 on Jul 17, 2007 when FBS rose to 105! After three months (on Oct 15, 2007) FBS & PPBS were 94 & 104.
- On the strength of three months' LOW NORMAL BLOOD SUGAR values, again decided to introduce sugar & fruit intake.
Apple & Orange
- 8. It was at this point of time that my wife (who is a voracious reader of women's periodicals in malayalam) showed a health article authored by a modern medicine practitioner. It categorically stated that low sweet fruits like APPLE & ORANGE are very safe & appropriate for even Diabetic patients. Additionally she reminded me the age old proverb "an apple a day keeps the doctor away". From next day onwards we ate either orange or apple daily; one each if small and half each if large. As I was under the impression that I am cured, tested only FBS for the next six months. All the FBS results were good. On Apr 29, 2008 when I got both FBS & PPBS tested (to consult my cardiologist) I was taken aback totally. Even though FBS was only 100, PPBS was very high - a shocking 165 mg/dl. Next test after ten weeks (Jul 08, 2008) clearly showed that things are worsening; FBS was 107 & PPBS was 192 - almost back to square one!
I said to myself hell with the expert's write-up on apple/orange. Those who coined the APPLE proverb would not have had Diabetes. Obviously I reverted to NO SWEET. Next test after six weeks (on Aug 22, 2008) showed FBS 100 & PPBS 135. Test on eight weeks (on Oct 17, 2008) marked a marginal increase in FBS to 109, PPBS dropped to 125 (both to within limits). Next test on Dec 20, 2008 (after 9 weeks) also pegged decrease; FBS to 95 & PPBS to 120.
- 9. Unfortunately a fourth time too I was misled by the low BS values & obviously restarted sugar - sweet intake and ended up in PPBS shooting up, to 161mg%.
- 10. It was at this time that I got HbA1C tested for the first time. The value was 6.3%. Then on things made a quantum change. Progress was in leaps & bounds. Rest is history.
Qualification test of PANCREAS
On July 8, 2009 associated with his visit to 10 houses between 6pm & 1030pm he consumed nearly a dozen sweet items. Next noon he took a wedding feast lunch. It was sumptuous with four (4) very sweet paayasams (porridge) and three (3) other sweet items in addition to more than a dozen other servings. (That day's breakfast was normal since long wheat mash had already been discontinued long ago, presuming cure.) A blood test after two hours of lunch showed a PPBS level of 100mg%!!
Time table
Event | Date | MM-DD-YY | Duration |
---|---|---|---|
Diet & food suppliment start | Jul 8, 2006 | 7-8-6 | |
FBS & PPBS within limits | Sep 1, 2006 | 9-1-6 | |
Time taken for FBS & PPBS within limits | 55 days | ||
Food suppliment (long wheat mash) intermittent discontinuites | Jul 2007 | ||
Time taken for Food suppliment intermittent breaks (discontinuities) | 1 year | ||
PPBS normal after heavy sweet intake | Jul 9, 2009 | 7-9-9 | |
Time taken for PPBS normal after heavy sweet intake | 3 years | ||
First proof test (GTT) | Sep 10, 2009 | 9-10-9 | 3Yr 2Months |
Second proof test (GTT at an NABH accredited lab) | Sep 14, 2009 | 9-14-9 | 3Yr 2Months |
Hindsight
- Had the diet relaxations been not done, the cure would have been faster and complete cure would have occurred much earlier.
- Had the information about HbA1C test been put to use earlier, misinterpreting low/normal FBS-PPBS values to be the indication of a cure would not have happened.
- But for the misinterpretation of low FBS-PPBS, the diet relaxation would not have been resorted to.
Anticipation
For patients on tablets or injection (insulin) time required for complete cure can be longer. This is because their treatment will have two phases:
- Tapering down the medicine dosage to zero.
- Allowing the system to repair the impaired pancreas (beta cells).
Future requirement[edit | edit source]
In order to arrive at more accurate time estimates, co-operation of more volunteers are required. Georgedappilly cannot harness his own body for the simple reason that he is no more a diabetic. Cooperation of diabetic persons who are on tablets or insulin injection for a fairly long period only will help arrive at better time estimates.
The long and emmer wheats
Long wheat
Long wheat is the trade name of a variety of wheat available in the Indian market. It is more than twice as costly as the common wheat (in India). A picture of the long wheat is given in the gallery below. It compares well with the Emmer wheat
A research scholar of Dept. of Botany, University of Kerala Campus, Kariavattom, Trivandrum clarified that the Botanical name of the long wheat is Triticum dicoccon.
Name in different languages[edit | edit source]
Sl.# | Language | Region | Country | Species | genus | Source |
---|---|---|---|---|---|---|
01 | Latin(Scientific) | Botanical | Many countries | T.dicoccon
(alias dicoccum) |
triticum | Internet |
02 | English | Global | Several countries | Emmer | Wheat | Wikipedia |
03 | English | India | India | Long | wheat | Traders |
04 | MalayalaM | Keralam | India | Soochi | goothamb (script) | Thandamma Mathew (Mother of georgedappilly) |
05 | MalayalaM | Keralam | India | Sooji | goothamb (verbal) | Thandamma Mathew (Mother of georgedappilly) |
06 | Tamil | Tamil Nadu | India | Samba | godhumai | A friend (Tamil)
k |
07 | Tamil | Tamil Nadu | India | Chamba | godhumai (alias) | An ex-colleague (Tamil) |
08 | Tamil | Nagercoil TN | India | Oosi | godhumai (alias) | An ex-neighbour (Tamil) |
09 | Kannada | Karnataka | India | Java | godhi | P.Janardanan |
10 | Goa | India | Suve | gov | U.Janardana Naik,VSSC | |
11 | Telugu | Andhra | India | Pedda | Godhumalu | S.Ramakrishna |
11a | Telugu | Aandhra | India | Poduvu | Godhumalu | https://healthunlocked.com/user/venkataramana |
11b | Telugu | Hyderabad, Erstwhile Andhra | India | Makka | Gehun | kashigari |
12 | Marathi | Maharashtra | India | Khapli | gahu | Veena Dhekane |
13 | Marathi | Maharashtra(Rural) | India | Lal | gahu | Chandan Prayag |
13a | Marathi | Pune | India | Jaada | Gehun | Anil Gupta Long Wheat interesting information; Anil Gupta Hindi speaking Rajastani traders of Pune |
13b | Marathi | Solapur Maharashtra | India | Jad | Gehun | Saxenaen Amit Photos of Long Wheat And Availability |
14 | Assaamese | Assaam | India | Suji | ghaum | Bhattacharya (RVTI Kzm) |
15 | Bengali | West Bengal | India | Suji | ghav | Rajan Panicker (P&P Kzm) |
16 | Hindi | Delhi, U P, Bihar,
M P, H P, Haryana |
India | Jau | ghehnoo | R.N.Singh,VSSC |
16A | Hindi | Central India | India | Lambe baal waala (long haired) | Gehu (Wheat) | Gphilip, DIHU |
17 | Italy | Italy | farro | Ashok Menon Malasia http://ucanr.edu/sites/Grown_in_Marin/Grown_In_Marin_News/GIM_News/Finding_the_right_rotation/Emmer_Wheat/ | ||
18 | Punjabi | Punjab | India | Javi | Kanak | https://healthunlocked.com/user/satishgoel |
19 | Bengali | West Bengal | India | Yob?? (Job ??) * | gom | A K Ray (VSSC) {* Yob=Written, Job=Spoken} |
19a | Bengali | West Bengal | India | Lal | Gam | G G Bandyopadhyay, VSSC |
20 | Punjabi | Punjab | India | Kanak | https://healthunlocked.com/user/634shy | |
21 | ?????? | Bangalore | India | Samba | Wheat | Nilgiri's https://healthunlocked.com/user/venkataramana |
22 | ????? | Punjab, Haryana and Rajasthan | India | Javi | Kanak | https://healthunlocked.com/user/satishgoel writes: Note: there are two types of javi, One like barley(Jau), and other Like Wheat(Kanak). Language= Punjab, Haryana and Rajasthan |
23 | Gujarati | Gujarat | India | Jouv | Ghau | Jayesh, Businessman, Chalai Bazaar, TVM |
23a | Gujarati | Gujarat | India | Bliya | avrs, member, healthunlocked BLIYA | |
24 | Kannada | Karnataka | India | ZAVE | GODHI | dssureshbabu Bangalore |
25 | ????? | Madhya Pradesh | India | Doudkhani | Wheat | Gospi I think LONG WHEAT is "Doudkhani" wheat of M.P. |
26 | Kashmiri (?) | Jammu & Kashmir | India | Sharbathi | Knak (or Kanak) | K L H through M N G Elayathu (both from VSSC) |
Emmer wheat
The Wikipedia says
- Emmer wheat belongs to Kingdom: Plantae, Order: Poales, Family: Poaceae, Genus: Triticum, Species: T.dicoccum, Binomial Name: Triticum dicoccum schrank, Synonyms: T.dicoccon, T.turgidum subsp.dicoccon
- Emmer is a hulled (husked) tetraploid wheat cultivated in mountainous areas. It is famous for making bread good in taste & texture standards. It has more fiber than common wheat.
Preparing the food supplement "suuji goothamb kanji" alias long wheat mash[edit | edit source]
Soaking Long-wheat (or any dry grain/seed) in water overnight makes cooking faster and saves precious fuel/energy.
- Measure out required quantity of whole-grain Long Wheat. Clean it by removing dust, husk & other foreign elements like stones etc.
Recipe (algorithm) one and DOSAGE
- My wife used to cook it without soaking in water & quantity sufficient for two meals together and retaining the second half in the cooking vessel itself as it will be sterile enough to preserve for nearly 12 hours. We used water equal to six times the volume of grain, to cook. Pressure regulating weight (PRW)is put in place when steam exit is continuous. At the first whistle flame is reduced to minimum/sim. Flame is put off at 45 minutes from putting the PRW in place. PRW is removed, to open the lid, only after sufficient time required for the pressure to come to normal level.
- On curing my Diabetes a word spread around & some interested patients ventured on LWMDR. Soon came complaints about difficulty in chewing & doubts about the quantity to be consumed.
- Chewing issue was solved by whipping/mashing in kitchen machine.
- With regard to quantity, a first cut thumb rule given was to use as much as required to quench the hunger or to fill the stomach.
- Later I took the measured quantity my wife used to cook for me and weighed it. That came around 65 grams. Soon I noticed a connection between this number & my body-weight - it was around 63 kilograms those days. Then I coined an easy to remember formula to specify the quantity of dry grain to cook the staple part of one meal of LWM.
- The quantity of Dry grain Long Wheat to be used to cook the staple part of one meal of LWM for a Diabetic patient is one gram (or more if required) per every kilogram of body-weigh of that patient. If stomach gets over-filled, reduce side dishes but not LWM.
- Do not drain out residual water after cooking; it also will contain essential nutrients - as in the case of many other decoctions. In case of difficulty or taste constraints, try using less water for cooking or drink the residual water later on conveniently; I repeat: never drain out residual water.
Click picture to enlarge it.
Recipe (algorithm) two
- Add double volume of water to the measured & cleaned Long Wheat and keep it covered in a container/vessel for soaking. Four-five hours of soaking is sufficient; keeping over-night is harmless.
- Whip in a mixie or grind in a kitchen machine or on a grinding stone, the soaked grain as a coarse or fine batter.
- Add sufficient (5-6 times the volume of dry grain equivalent of) water & boil preferably in an open vessel till cooked. Stir continuously to avoid uneven cooking.
- Add salt (if permitted, that is if there is no BP or Albumin complaint). It will resemble cooked OATS.
- Consume with permitted side dishes (Veg/Non-veg excluding sweet, starchy & root vegetables).
- This recipe makes cooking much faster, consumption & digestion easier.
- As in the case of other things told here, I have personally experienced/verified this process & product.
- Concistancy (thick/loose) of the food can be adjusted by varying the following parameters:
- Duration of soaking in water between one and four hours,
- Duration & speed of whipping/wet-grinding,
- Time of adding water during whipping/wet-grinding & quantity of water added.
Click picture to enlarge it.
Recipe (algorithm) three (time optimised two)
- Add double volume of water to the measured & cleaned Long Wheat and heat it in the cooking vessel for a minute or two.
- Keep it covered in the same container/vessel for 5-10 minutes for soaking. (5 mins is sufficient for batter-bits combination).
- Whip in a mixie or grind in a kitchen machine or on a grinding stone, the soaked grain along with the water as a coarse or fine batter.
- Use sufficient (5-6 times the volume of dry grain equivalent of) water to extract the residual batter sticking on the inner surface of the machine by rinsing & use the same to dilute the batter.
- Boil in an open vessel till cooked. Stir continuously to avoid uneven cooking.
- In 3-4 minutes on stove, consistency of the stuff changes. Then heating may be lowered to minimum. In another 6-7 minutes once more consistency may change indicating cooking completion. Bubbling in the cooking vessel on flame with slower stirring also indicates cooking completion.
Rest, given below, is same as in recipe two.
- Add salt (if permitted, that is if there is no BP or Albumin complaint). It will resemble cooked OATS.
- Consume with permitted side dishes (Veg/Non-veg excluding sweet, starchy & root vegetables).
- This recipe makes cooking still faster than recipe-2, consumption & digestion as in the case of recipe-2.
- As in the case of other things told here, I have personally experienced/verified this process & product.
- Concistancy (thick/loose) of the food can be adjusted by varying the following parameters:
- Duration of soaking in hot water between five and ten minutes,
- Duration & speed of whipping/wet-grinding,
- Time of adding water during whipping/wet-grinding & quantity of water added.
Recipe four - unleavened bread using Long Wheat flour
Unleavened bread is nothing but the Indian Chappaatthi. Hence narrating preparation procedure is skipped.
Scientific evidence corroborating Georgedappilly's claim (2009) on hypoglycemic efficacy ofLong Wheat had been lying dormant for one and a half decades in a Scientific Paper [2]. Please note the positive fact contained in the included data (TAble II of the paper) [2].
The point that authors left unnoticed in the paper & caught my attention as very important is: the increase in blood sugar that happened due to the experimental diet in two fortnights was reverted by the next one fortnight in the case of Group I (who used Dicoccum whole Wheat flour) but not so in the case of Group II (who used Bread Wheat flour)!!!
This study indicates that even LW flour in the form of unleavened bread works (not only mash!) in regulating the Blood Sugar of Diabetes patients.
The referred paper is certainly a great encouragement to all those who are on LWMDR as well as those who are contemplating on trying LWMDR.
They (experts) say
Diabetes India Association[edit | edit source]
- Diabetes India Association says "The 1997 WHO report has shown that there is a marked increase in the number of people affected with diabetes and this trend is scheduled to grow in geometric proportions in the next couple of decades".
YEAR | NO OF PEOPLE AFFECTED
(in millions) |
---|---|
1995 | 124.7 |
2000 | 153.9 |
2025 | 299.1 |
- DIA President cautions against child diabetes explosion.
- 'The discovery of insulin was the greatest boon to the children with diabetes. To tell them the story of the discovery, a comic strip was printed in the early twenties. Here is the comic strip
[edit | edit source]
- Diabetes India Association (-> open house) in different articles talk about a few related issues.
- The DIA says "the 1997 WHO report has shown that there is a marked increase in the number of people affected with diabetes and this trend is scheduled to grow in geometric proportions in the next couple of decades".
- The prevalence of Type 2 diabetes (non insulin dependent diabetes, NIDDM) is increasing all over the world. THE DIABETES BOMB...........READY TO EXPLODE. In India the prevalence of diabetes has increased from 5.2% in 1983 to 8.2% in 1989 and 11.6% in 1995. With the rising trend in the prevalence of diabetes, it is estimated that by the year 2000 A.D. the number of diabetic persons in India will be 33 million and the prevalence will be 14.7% in subjects aged 20 years or more. The World Health Organization (WHO) has recently acknowledged that India has the maximum number of diabetic patients in any given country in the year 1995 (19 million) and that this would increase to 57 million by the year 2025. India has thus become the "Diabetic Capital of the World".
- The epidemic of diabetes is so widespread that there would be scarcely be anyone who does not know someone with diabetes.....
American Diabetes Association[edit | edit source]
- The Living with diabetes page of ADA says 'they provide information and resources to help every child and every family adjust to life with diabetes'
- More links are available -> there.
Wikipedia[edit | edit source]
- 'Diabetes mellitus type 2 is a chronic, progressive disease that has no established cure......' says Wikipedia.
- The words 'no definitive cure for Type 2 diabetes' can be seen here.
My Diabetes has not yet returned
Last Wednesday (27.01.2010) I had the regular quarterly health check up. Along with Lipid profile etc, blood sugar also was monitored. Fasting Blood Sugar (FBS) was 100 mg%, Post Prandial Blood Sugar (PPBS) showed 105 mg% & Glycosylated Haemoglobin (HbA1C) was 6%.
-
Blood sugar (FBS, PPBS & HbA1C) lab result printout after seven months of cure
-
Blood sugar graph(FBS & PPBS) including result of 27 jan 2010 (ie after seven months of cure)
-
On the way to the consulting room of my physician, I met a friend & his wife. She is diabetic. Just 2 Idlis & ½ a cup of tea took her PPBS to 195 mg% where as 3 Idlis & a full cup of coffee took my PPBS to 105 mg% only!
What is happening in my case is entirely different from what many people anticipated &/or told on hearing my declaration of the cure of my diabetes.
In spite of taking rice based food thrice daily, coffee/tea with sugar, bakery sweets after lunch frequently (not daily) & 5-6 plantain fruits most of the days for seven (7) months, my diabetes DIDN'T RETURN.
What most of the visitors of my poster presentation (see albums of day 1 of 3, day 2 of 3 & day 3 of 3) (at the KERALA GRAMAM (Kerala village) exhibition arranged in connection with the 97th Indian Science Congress (ISC2010) at the Kerala University Campus, Kariavattom, Trivandrum) wanted to know was "WHAT IS THE GUARANTEE (OR ARE YOU SURE) THAT DIABETES WILL NOT RETURN?"
My reply was: "AT THIS POINT OF TIME WHAT I CAN SAY IS THAT IN SPITE OF NORMAL DIET MY DIABETES DID NOT RETURN FOR SIX (6) MONTHS & IF YOU ASK ME AFTER ANOTHER MONTH, I WILL SAY THAT MY DIABETES DID NOT RETURN FOR SEVEN (7) MONTHS". This has come true on Jan 27, 2010. Only after the elapse of one year (5 more months from now), I can make a statement that my diabetes did not return for an year.
In order to make a general statement that diabetes will not return for any specific duration, more time, cases & case studies are required. If the present trend continues, that may not happen in the near future for sure. Whom ever I encounter turns out to be unfortunately a pessimist.
One of my friends (a blogger reffered as Ankil) was more eager to ascertain that my claim of cure of diabetes is WRONG, than positively responding to my offer for trying to explore the possibility of a cure of HIS OWN DECADES OLD DIABETES.
Alas!!!
I am reminded of the anecdote of KERALA CRABS.
35 weeks!
Today (Tue, March 9, 2010) I took the monthly blood sugar test (GOD-POD method). It shows FBS 80 mg% & PPBS 95mg%. This marks the successful completion of consecutive 8 months of the victory of my Pancreas over Diabetes!
Again Praise The Lord!!
But unfortunately even people with decades old diabetes, having progressive & degenerating experience finds disbelieving these results more comfortable.
One more month of no return: 39 weeks now!
The blood sugar test results of April 10, 2010 confirms that my diabetes has not returned even after 39 weeks of normal diet containing starch & sugar. FBS is 90 mg% and PPBS is 110 mg%. These values are well within limits. My pancreas is still going strong. Jump back to Section 2 - (My diabetes has not yet returned). File:DiabCureHistoryGraphEasyplot.ppt
Second anniversary special blood test over 24 hours
On completion of two years of CURE of my diabetes, a different sequence of blood sugar tests were carried out and the results are given below (thanks to my daughters for the glucometer & camera which made the total process very handy). As is customary, tested FBS to begin with. It was 92 mg/dl at 7:26 AM. Next, tested morning-PPBS at 2 hours of breakfast. It was 136 mg/dl. Noon-PPBS at 2 hours of lunch was 135 mg/dl & night-PPBS at 2 hours of supper was 133 mg/dl at 9:44 PM. Next morning it was felt that one more FBS test would give a completeness to the picture. Hence it was done & got 84 mg/dl at 7:39 AM. This special sequence test re-confirmes the fact that my experiences & findings on Diabetes CURE was & is really REAL since July 2009. It is indeed very heartening to note that my experience and findings of July 2009 gets corroborated by the study results of Dr. Roy Taylor of the prestigeous Newcastle University, UK.
Date | Time | Test | Result
mg/dl |
---|---|---|---|
21.07.2011 | 0726 | F B S | 092 |
21.07.2011 | 1106 | P P B S | 136 |
21.07.2011 | 1542 | P P B S | 135 |
21.07.2011 | 2144 | P P B S | 133 |
22.07.2011 | 0739 | F B S | 084 |
142 weeks!
The blood sugar test results of April 03, 2012 confirms that my diabetes has not returned even after 142 weeks of normal diet containing starch, sugar & fat. FBS is 88 mg% and PPBS is 82 mg%. These values are well within limits. My pancreas is still going stronger!!.
My Diabetes is still not back (3+ years)
Feb 22,2013; FBS 100 mg/dl, PPBS 104 mg/dl, HbA1C 5.9%
Fourth anniversary of my diabetes cure
July 2009 - July 2013. HbA1c steady at 5.9% from Jan 2012 to April 2013 with normal rice diet, fruits & coffee/tea with sugar.
Lab results from July 2006[edit | edit source]
Oldest lab report located on 30.09.2018 in an old file
Numerical representation
Date | FBS(mg/dl) | PPBS(mg/dl) | HbA1c(%) | Remarks |
---|---|---|---|---|
27.09.2005 | 120 | .. | .. | Earliest report of Dr Ramachandran's Diagnostic Services, Kumarapuram 695011 located in old file on 30.09.2018 (see picture). |
07.07.2006 | 118 | 197 | .. | .. Not tested |
01.08.2006 | 120 | 155 | .. | Latest entry in RED |
01.09.2006 | 100 | 130 | .. | |
12.09.2006 | 100 | 155 | .. | |
04.10.2006 | 102 | 160 | .. | |
07.11.2006 | 121 | 166 | .. | |
02.01.2007 | 80 | 98 | .. | |
03.02.2007 | 83 | 96 | .. | |
29.05.2007 | 85 | 155 | .. | |
17.07.2007 | 105 | 100 | .. | |
15.10.2007 | 94 | 104 | .. | |
29.04.2008 | 100 | 165 | .. | |
08.07.2008 | 107 | 192 | .. | |
20.01.2009 | 108 ## | 161 ## | 6.3* | *24.01.2009 |
22.04.2009 | 115 ## | 74 ## | 6.1 | |
......Till this | Diabetic | .......After this | NOT Diabetic | Hence SPECIAL regimen DISCONTINUED |
09.07.2009 | .. | 100# | .. | #6 Sweets |
25.07.2009 | 108 ## | 74 ## | 5.9 | |
10.09.2009 | 85 | 100$ | .. | $ GTT-1 |
14.09.2009 | 109 | 118$$ | .. | $$ GTT-2 |
21.12.2009 | .. | 112 | .. | |
07.10.2009 | 97 ## | 98 ## | 6.1 | |
27.01.2010 | 100 ## | 105 ## | 6.0 | |
09.03.2010 | 80 | 95 | .. | |
10.04.2010 | 90 | 110 | .. | |
28.04.2010 | 105 ## | 131 ## | 6.2 | |
26.05.2010 | 80 | 100 | .. | |
03.07.2010 | 95 | 110 | .. | |
31.07.2010 | 107 ## | 88 ## | 6.2 | |
27.08.2010 | .. | 127 | .. | |
03.09.2010 | 80 | 140 | .. | |
09.10.2010 | 80 | 90 | .. | |
05.11.2010 | 90 GP | 100 GP | .. | GP GOD-POD method |
07.12.2010 | 96 ## | 94 ## | 5.9 | ## Hexokinase method |
17.02.2011 | 85 | .. | .. | |
18.02.2011 | .. | 130 | .. | |
25.03.2011 | 97 | 145 | 6.1 | Serum Insulin (Eclia) 10.03 micro U/ml
Ref. Range: 2.6 - 24.9 |
16.04.2011 | 95 | 140 | .. | |
27.05.2011 | 90 GM | .. | GM Glucometer | |
03.06.2011 | 85 GM | .. | ||
10.06.2011 | 86 GM | .. | ||
13.06.2011 | 85 GP | 100 GP | .. | |
12.07.2011 | 104 ## | 99 ## | 6.2 | .. |
13.08.2011 | 75 GP | 90 GP | .. | .. |
13.09.2011 | .. | 105 | .. | .. |
14.10.2011 | 89 | 92 | 6.3 | .. |
15.11.2011 | 90 (19.11.2011) | 119 | .. | PPBS with three sweets |
27.12.2011 | 96 (28.12.2011) GM | 93 GM | .. | PPBS lunch |
13.01.2012 | 100 ## | 85 ## | 5.9 | Children vacation, X'mas, Newyear |
17.02.2012 | 93 GM | 118 GM | .. | |
08-03-2012 | 85 GM | 127(16-03-2012) GM | .. | |
22-03-2012 | 88 GM | 82(27-03-2012) | .. | PPBS lunch |
29.03.2012 | 96 GM | 125 GM$$$ | .. | $$$ GTT-3 |
29-08-2012 | 88 GM | 137 GM | .. | Bedridden due to back pain, fools day onwards |
12.10.2012 | 99 GM | 134 GM | .. | Back on legs |
09.11.2012 | 104## | 113## | 5.9 | Test at ACHSI and NABL certified lab |
02.01.2013 | 89 GM | 132 GM | .. | |
19.01.2013 | 91 GM | .. | .. | 1/2/3 coffee/tea with milk&sugar in addition to bf, lunch & dinner regularly |
22.02.2013 | 100 ## | 104 ## | 5.9 | PPBS after BF of {3 Iddali (alias Idli) = 300 k cal + 1 cup daal saambaar + 0.5 cup coconut chutney + coffee with milk & sugar} |
21.03.2013 | 99 GM | .. | .. | |
17.04.2013 | 92 GM-K | 131 GM-KL | 5.9 | K=@KIMS Hospital on 16Apr. L=Lunch at 330pm, test at 530pm |
07.05.2013 | 94 GMK | .. | K KIMS Hospital | |
04.06.2013 | 99 | .. | .. | |
25.06.2013 | .. | 109 GM-S | .. | S=Supper at 840pm, test at 1040pm |
07.08.2013 | 99 ## | .. | 5.6 | Routine quarterly check up |
11.08.2013 | 90 GM | 124 GM | .. | Home Gluco Meter |
04.09.2013 | .. | 117 GM & | .. | & Supper PP on full-veg-feast-lunch-with-four-desserts-and-a-fruit-day |
30.09.2013 | 97 GM | .. | .. | 12.5 Hrs Fasting |
08.10.2013 | 87 GM (9.X.13) | 116 GM | .. | .. |
01.11.2013 | 92 GM | .. | .. | .. |
06.11.2013 | 103 ## | .. | 5.9 | .. |
09.11.2013 | .. | 130 GM | .. | .. |
22.11.2013 | 93 GM (1)
94 GM (2) |
.. | .. | (1) 9Hr 45 min FASTING
(2) 14 HR FASTING |
30.11.2013 | .. | 127 GM | .. | |
05.02.2014 | 99 ## | .. | 5.8 | |
28.03.2014 | 95 GM | 104 GM | .. | After lunch with cooked rice (300 gm) & side dishes; BF also rice based. |
06.05.2014 | 95 GM, 98 ## | .. | 5.9 | GM Glucometer, ## Hexokinase method |
29.05.2014 | 89 GM | 131 GM | .. | |
06.11.2014 | 5.9 | .. | ||
27.12.2014 | 90@ | 143@ | .. | @ Panchakarma hospital, Poojappura, Trivandrum |
06.02.2015 | 105 ## | .. | 6.0 | .. |
13.04.2015 | 82 GP | 148 GP | .. | .. |
Booster | dose of LWM | (one meal daily) | started | |
24.04.2015 | 91 ## | .. | 6.0 | .. |
23.06.2015 | 82 GP | 124 GP | .. | .. |
06.08.2015 | 95 ## | .. | 6.2 HPLC | .. |
27.08.2015 | 86 GP | 119 &## | .. | & 28.08.2015 |
09.09.2015 | .. | 104 GP | .. | .. |
22.10.2015 | .. | 108 GP | .. | .. |
30.09.2015 | .. | .. | 5.7 ! | ! Colorimetry |
03.11.2015 | 91 ## | 90 ## | 6.0 | .. |
17.11.2015 | .. | 108 ## | .. | .. |
28.11.2015 | 73 GP | .. | .. | .. |
18.12.2015 | .. | 81 | .. | .. |
28.05.2016 | 90 GP | 73 GP | .. | BF: 3 appam of rice dough + egg curry + coffee with milk & sugar |
2016 Onward (Numerical repreentation) continued
Date | FBS(mg/dl) | PPBS(mg/dl) | HbA1c(%) | IR2 | Remarks |
---|---|---|---|---|---|
28.05.2016 | 90 GP | 73 GP | .. | .. Not tested. Breakfast: 3 Appam of rice douh + 1 egg curry + Coffee with Milk & Sugar | |
03.08.2016 | 95 | 86 | .. | Usual breakfast 3 idlis + Coconut Chutney + Saambaar + Coffee with Sugar & Milk.
Serum Insulin (ECLIA) 5.41 micro U/ml (Range 2.60 - 24.90) | |
23.09.2016 | .. | 68 | 5.00 | BF 6 slices of Sweet Rusk of "modern" bakeries + a large Banana fruit (hard type, very sweet), a heavy BF. | |
01.11.2016 | 99 | .. | 5.9 | 0.924 | |
04.02.2017 | 96 | 85 GP | .. | 1.104 | |
12.04.2017 | 90 | 92 | .. | PPBS on April 14 at 2hrs after BF: Upma + plantain fruit + 1 glass milk with 2 spoons of sugar. | |
01.06.2017 | 94 | .. | .. | 1.051 | |
26.08.2017 | 94 | .. | 5.8 | 1.260 | Routine quarterly cardiology checkup. Usual 3 rice meals daily, coffee/tea with sugar, fruits almost frequently & no diab medication, no hard exercise. |
28.11.2017 | 97 | .. | .. | 0.979 | " " " IR2=Insulin Resistance (2nd formula) desirable value LE (<=) 2.0 |
31.05.2018 | 95 ## | 94 GP | 5.6 | 1.0188 | ## > Hexokinase
GP > GOD-POD |
26.11.2020 | 91 | 5.8 | 0.744 | KIMS Home Lab Service | |
23.8.2021 | 88 | 5.8 | 0.954 | KIMS Home Lab Service | |
Lab report copy (not latest)
Graphical representation
(Click picture to enlarge it. Right click to enlarge in another window)
2006 to 2012.................. and 2012 Onwards
Some of my other patients
Patient code: SDPN.M.P.T.K.I.
- Sex: Male
- Age: 59 (on May 2010)
- Diabetes diagnosed: 1990
- Lab results from April 2010
Numerical representation
Date | FBS(mg/dl) | PPBS(mg/dl) | HbA1c(%) | Tabs | Remarks |
---|---|---|---|---|---|
04.04.2010 | 207 | 288 | .. | ??-??-?? | .. Not tested |
30.04.2010 | 110 | .. | 10.0 | 1.0 - 0.0 - 1.0 | Latest entry in RED |
07.05.2010 | 80 | .. | .. | 1.0 - 0.0 - 0.5 | |
17.05.2010 | 101 | .. | .. | 1.0 - 0.0 - 0.5 | |
03.06.2010 | 96 | .. | .. | 1.0 - 0.0 - 0.0 | |
17.06.2010 | 125 | .. | .. | 0.0 - 0.0 - 0.0 | |
25.06.2010 | 119 | .. | .. | 0.0 - 0.0 - 0.0 | |
03.07.2010 | 114 | .. | .. | 0.0 - 0.0 - 0.0 | |
27.07.2010 | 109 | .. | .. | 0.0 - 0.0 - 0.0 | |
30.07.2010 | .. | .. | 7.0 | 0.0 - 0.0 - 0.0 | |
24.08.2010 | .. | 120 | .. | 0.0 - 0.0 - 0.0 | |
05.10.2010 | 145 | .. | 7.0 | 0.0 - 0.0 - 0.0 | |
09.12.2010 | 137 | .. | 8.6 | 0.0 - 0.0 - 0.0 | |
10.12.2010 | .. | 111 | .. | 0.0 - 0.0 - 0.0 | |
23.02.2011 | 120 | .. | 6.6 | 0.0 - 0.0 - 0.0 | |
11.04.2011 | 134 | .. | .. | 0.0 - 0.0 - 0.0 | |
02.06.2011 | .. | 105 | 6.4 | 0.0 - 0.0 - 0.0 | .. |
09.08.2011 | .. | 185 | 7.5 | 0.0 - 0.0 - 0.0 | |
17.08.2011 | .. | 140 | .. | 0.0 - 0.0 - 0.0 | |
26.08.2011 | .. | 151 | .. | 0.0 - 0.0 - 0.0 | |
14.11.2011 | .. | 114 | .. | 0.0 - 0.0 - 0.0 | |
06.01.2012 | .. | 97 | 6.95 | 0.0 - 0.0 - 0.0 | |
12.01.2012 | 113 | .. | .. | 0.0 - 0.0 - 0.0 | |
31.01.2012 | .. | 97 | .. | 0.0 - 0.0 - 0.0 | |
.. | .. |
Patient code: 41SKLM.F.M.T.K.I.
- Sex: F
- Age: 70 as on May 2010
- Diab diagnosed: 2000
- Lab results from May 2010
Numerical representation
Date | FBS(mg/dl) | PPBS(mg/dl) | HbA1c(%) | Tabs | Remarks |
---|---|---|---|---|---|
10-05-10 | 110 | 140 | .. | 1.0 - 0.0 - 0.5 | .. Not tested |
21-05-10 | 137 | 10.0 | 1.0 - 0.0 - 0.5 | Latest entry in RED | |
01-06-10 | 125 | .. | 1.0 - 0.0 - 0.5 | ||
10-06-10 | 110 | .. | 1.0 - 0.0 - 0.0 | ||
14-07-10 | 137 | .. | 1.0 - 0.0 - 0.5 | ||
28-07-10 | 140 | .. | 1.0 - 0.0 - 0.5 | ||
17-08-10 | 120 | .. | 1.0 - 0.0 - 0.0 | ||
01-09-10 | 100 | .. | 0.5 - 0.0 - 0.0 | ||
17-09-10 | 110 | .. | 0.0 - 0.0 - 0.0 | ||
28-09-10 | 109 | 150 | 7.0 | 0.0 - 0.0 - 0.0 | |
16-02-11 | 159 | .. | 0.0 - 0.0 - 0.0 | ||
20-02-11 | 134 | 7.0 | 0.0 - 0.0 - 0.0 | ||
18-04-11 | 215 | 8.6 | 0.0 - 0.0 - 0.0 | ||
28-04-11 | 227 | 0.0 - 0.0 - 0.0 | |||
26-05-11 | 120 | .. | 0.0 - 0.0 - 0.0 | ||
13-06-11 | 160 | 6.6 | 0.0 - 0.0 - 0.0 | ||
04-07-11 | 108 | .. | 0.0 - 0.0 - 0.0 | ||
12-07-11 | 113 | 6.4 | 0.0 - 0.0 - 0.0 | .. | |
04-08-11 | 112 | 7.5 | 0.0 - 0.0 - 0.0 | ||
16-08-11 | 121 | .. | 0.0 - 0.0 - 0.0 | ||
16-09-11 | 130 | .. | 0.0 - 0.0 - 0.0 | ||
22-10-11 | 212 | .. | 0.0 - 0.0 - 0.0 | ||
31-10-11 | 184 | 6.95 | 0.0 - 0.0 - 0.0 | ||
11-11-11 | 214 | .. | 0.0 - 0.0 - 0.0 | ||
13-11-11 | 136 | 0.0 - 0.0 - 0.0 | |||
17-12-11 | 150 | 0.0 - 0.0 - 0.0 | |||
01-02-12 | .. | 128 | .. | 0.0 - 0.0 - 0.0 | |
10-02-12 | 118 | .. | .. | 0.0 - 0.0 - 0.0 | |
13-03-12 | 120 | .. | .. | 0.0 - 0.0 - 0.0 | Start 1 Plantain (small variety banana)
in empty stomach |
24-03-12 | 132 | .. | .. | 0.0 - 0.0 - 0.0 | Stop plantain, start exercise |
03-05-12 | 109 | .. | .. | 0.0 - 0.0 - 0.0 | |
30-05-12 | 100 | 140 | .. | 0.0 - 0.0 - 0.0 | Exrc^ 22# |
12-09-12 | .. | 168 | .. | 0.0 - 0.0 - 0.0 | |
01-11-12 | .. | 130 | .. | 0.0 - 0.0 - 0.0 | PP Lunch |
01-12-12 | 122 | .. | .. | 0.0 - 0.0 - 0.0 | Impaired Fasting Glucose? Patient next to cure?
Confirm with PPBS next time. |
04-01-13 | 105 | .. | .. | 0.0 - 0.0 - 0.0 | |
04-02-13 | 112 | .. | .. | 0.0 - 0.0 - 0.0 | |
10-02-13 | 132 GOD-POD | .. | .. | 0.0 - 0.0 - 0.0 | Impaired Fasting Glucose? Confirm with PPBS next time. |
14-02-13 | .. | 137 GOD-POD | .. | 0.0 - 0.0 - 0.0 | It was Hepatic Insulin Impairment. |
04-10-13 | .. | 115 GOD-POD | .. | 0.0 - 0.0 - 0.0 | Very close to cure. 2b confirmed with HbA1c |
.. | .. |
Patient code: AshMnnOthn.-.-.-.KL.Mlsa.
- Sex: M, Ht 174 cm (5' 8.5"), Wt 80 kg (176.4 lbs), BMI 26.4
- Age: 60 as on Feb 2013
- Diab diagnosed: 1991
- Lab results etc from xxx 1991
Numerical representation[edit | edit source]
Date | FBS(mmol/L){mg/dL} | PPBS(mmol/L){mg/dL} | HbA1c(%) | Tabs/Insulin | Remarks.
Latest entry in RED |
---|---|---|---|---|---|
xx-xx-91 | ?? | ?? | ?? | *1.0 - *1.0 - *1.0
.**1. - **0.0 - **1.0 |
* T.Metformin 500 mg
.** T.Diamicron 80 mg |
xx-xx-99 | (8) | (18) | xx | $6.0 - $0.0 - $6.0
.*1.0 - *1.0 - *1.0 |
$ I.Mixtard
.* T.Metformin 500 mg |
Insulin | Gradually | increasing | over the | years from 6-0-6 to | 35-00-25 |
xx-xx-09 | ?? | ?? | ?? | $35.0 - $0.0 - $25.0
.*1.0 - *1.0 - *1.0 |
$ I.Mixtard
.* T.Metformin 500 mg |
xx-xx-10 | ?? | ?? | ?? | $35.0 - $0.0 - $25.0
.*1.0 - *1.0 - *1.0 |
$ I.Novomix
.* T.Metformin 500 mg S |
xx-01-12 | ?? | ?? | 9.5 | Endocrynologist | Consultation Feb 2012 |
xx-02-12 | ?? | ?? | 9.5 | $27.0 - $37.0 - $35.0
.*1.0 - *0.0 - *1.0 |
$ I.Novomix
.* T.Metformin 850 mg |
29-11-12 | (6.5) | .. | 7.5 | $27.0 - $37.0 - $35.0
.*1.0 - *0.0 - *1.0 |
$ I.Novomix
.* T.Metformin 850 mg |
.. | .. |
More on User:Georgedappilly's unique experience
Read more on User:Georgedappilly's different diabetic experience & opinions at Posts as well as Activity of another public forum named HealthUnlocked/DiametesIndia.
It is felt that...[edit | edit source]
- No one else has ever accomplished this unique goal of curing Diabetes in human history till July 2009. It was considered to be humanly impossible. Hence it is felt that any one will be justified in opining that this is the Lords doing, It is marvelous in our eyes..
- Having conquored Diabetes which is, according to WHO, claiming six (6) lives every minute I wish to take the message and know how to as many victims in as short a time as possible because "no one after lighting a lamp puts it under the bushel basket, but on the lampstand, and it gives light to all in the house. In the same way, let your light shine before others, so that they may see your good works...".
- From the responses of a few responsible persons it is felt that they mistook these efforts to be an attempt to infringe upon their territory."Do not think that I have come to abolish the law or the prophets; I have come not to abolish but to fulfill."
- I desire to be a taste-maker, like salt, to our society. It vanishes itself into other components, improves their qualities & makes the final product tasty. The book named Bible that has the maximum number of copies printed in human history says: "You are the salt of the earth; but if salt has lost its taste, how can its saltiness be restored? It is no longer good for anything, but is thrown out and trampled under foot".
- ------------------------
- It is felt that many people mistake me for a Long Wheat cultivator, farmer, trader or researcher. They think that I am cunningly trying to promote the prospects of Long Wheat by misleading Diabetic patients, pretending to have cured Diabetes which is considered by modern science and medicine people as a syndrome & (hence) incurable. I pity them and feel that it is already explained/replied in a book written twenty centuries ago; passage is there.
- -----------------------Albert Einstein cannot be wrong...
- He said "if at first the idea is not absurd, then there is no hope for it".
George Bernard Shaw is right...
- He said: "if you have an apple and I have an apple and we exchange these apples then you and I will still each have an apple. But if you have an idea and I have an idea and we exchange these ideas, then each of us will have two ideas."
On inventions
Prof.A.W.Bickerton, on lunar mission, was wrong then
British Chemist (1926)
- "This foolish idea of shooting at the moon is an example of the absurd lengths to which vicious specialisation will carry scientists working in thought-tight compartments. To escape the earth's gravitation, a projectile needs a velocity of 7 Mps(*). The thermal energy at this speed is 15180 cals. Hence the proposition appears to be basically impossible."
(* M denotes miles)
Arthur C Clark replying Bickerton...
Clark's Law propounded by the writer(science fiction) and scientist (inventor of Geo-stationary orbit)
It states in effect that
- If a respected senior scientist says a thing can be done, he or she is almost always correct; if the scientist says it cannot be done, he or she is almost always wrong.
Another Breaking news of diabetes cure on June 24, 2011
A leading MalayalaM news paper (Malayala Manorama of Kerala - India) of today (25 June 2011) has reported an epoch making finding on curability of diabetes. It obviously is of great interest to User:Georgedappilly along with all the readers of this page and the billions of diabetics world over! A photograph of the news and its English translation are given below.
Translation
Title of the news: Diabetes likely to be cured by low calorie food.
Body of the news:
London: Researchers of Newcastle University claim that the Type-2 diabetes seen in elders can be completely cured by sticking to low calorie food pattern for two months continuously. What this finding rejects is the thinking that diabetes is a life long disease.
The Guardian news paper reported that the experiment was carried out in 11 diabetic patients. Daily they were given food having only 600 calories in the form of vegetables and juices for two months. Medical team subjected these ones to continuous examinations.
By one week itself the fasting blood sugar (FBS) reading became normal. It was found out that the extent of Fat in Pancreas was lowered and that the Pancreas was producing Insulin as it was before the onset of diabetes.
They were returned to normal diet after two months. It was found in later examinations that 7 patients were completely cured of the disease. This method is not useful for juvenile diabetes. It is the Type-2 of adults that is cured by food regulation.
This type of diabetes is caused when Insulin production is impaired by the malfunctioning of liver and pancreas when the fat from the excess food accumulate in them. Prof. Roy Taylor of Newcastle University explained that reinstating the insulin production on melting away the fat accumulated in the pancreas, by following low calorie food pattern is the methodology.
Original full paper[edit | edit source]
This link will take you to the full text of the original paper of Prof. Dr. Roy Teylor et al
English-language articles[edit | edit source]
This was also reported in English news sources, including:
- British docs say they can cure diabetes: How?, CBSNews.com, June 24, 2011
- Early Stage Diabetes Reversible With Two Month 600 Calorie Per Day Diet, Medical News Today, 24 Jun 2011
- Crash course diet reverses Type-2 diabetes in a week, Telegraph.co.uk, June 24, 2011
- Low-calorie diet offers hope of cure for type 2 diabetes , Oneclick.indiatimes.com, June 24, 2011
- Very Low-Calorie Diet May Reverse Diabetes, Webmed Diabetes Health Center.
Participants reported finding the extremely strict diet difficult, but the results are very positive.
Note that positive effects from exercise and diet have long been known in type-2 diabetes, but these articles suggest that actually curing the condition was not part of conventional medical thinking.
Note also that the the study appeared to focus on a particular form of diet. It remains to be studied what the success rates are for other forms of diet (different foods, caloric intake and lengths of time), such as George's diet.
Responses of User:Georgedappilly to various English language articles[edit | edit source]
- Comment 1: I am really excited to read this fantastic article because it validates my findings. From 2006 to 2009 I was Diabetic, but NOT afterwards. I am on normal diet afterwards. My experiences and blood-sugar test results are available at https://www.appropedia.org/Diabetes_mellitus_cured.One can reach it by a Google search of LONG WHEAT. It appears in the first page of search results itself.
A graphical representation of the results are also provided for ease of understanding. This article seems to be visited by more & more people. I am very much eager to discuss & share my experiences with fellow people for their benefit. E.M.George
- Comment 2: The claim of complete cure of diabetes in 8 weeks, of the Newcastle University research team appears to be somewhat unrealistic. Based on hands-on-experience of curing own diabetes & trying to cure that of more than 100 others, it is very strongly felt that two months is too short a time to cure diabetes completely. This is explained in detail in the "Pit-falls" section of my Appropedia article. It is indubitable that 2 months is more than sufficient to completely control diabetes, but not to cure. This is told because I have really gone through this phase, as explained in the said section "Pit-falls".
600 calories a day appears to be too low. The diet I followed seems to be much easier, comfortable as well as safer. My diet had much more calories a day. I keep telling my clients that even though the special regimen is simple, it is like swimming, driving or riding a bicycle. One has to learn it under an expert & unfortunately we don't have many experts. First of all one has to assess the initial conditions such as FBS (8 to 14 hours after any food), PPBS (AT 2 hours after a meal), HbA1C (Glycosylated haemoglobin) and if possible Fasting Serum Insulin. Additionally details regarding other illness, medication for management of Diabetes, body weight & height (or BMI value) etc also are to be considered. Me as well as many of my clients feel that it is rather a smooth sail. Even though it took three years for me to cure my diabetes (2006 t0 2009), it would have been cured in much shorter a time, had I not encountered the pit-falls explained in the Appropedia article with the help of a graphical representation of blood sugar values. The mention in another comment that starvation is a cure for all problems is not true. It is felt that strict 600 calorie-a-day veg. diet need not be a necessary & sufficient condition to cure diabetes.
- Comment 3: In fact the Newcastle research team & their 7 out of 11 volunteers seem to have fallen into the first pit in which I had fallen in early 2006. Nothing to worry. They will recognize it and come out soon! Aaaaand they will re-invent things.
- Comment 4: See Things In Totality. Compartmentalization & Over-specialization Do Harm, Mostly! posted by E.M.George on 28 Jun 2011 at 6:00 am
600 calories a day is at least impractical (AchsBuster) if not insane (Sonya)! There is an Indian proverb which means anything in excess is bad. So is starving or eating!! I was diabetic, but not now. I am on my usual diet since july 2009. The dreaded diabetes has not returned yet. Even if it returns, I am not afraid. I know how to cure it once again. One need not be on 600 cal a day, or no carb, or no meat diet, or keep running to cure diabetes. One has to know the limits & adhere to it. There is a food substitute mentioned in the ancient Indian medical literature (Aayurveeda) for curing the illness that expels sugar through urine. A judicious use of that grain (Long wheat) and avoidance of sugar & fruits, moderate control on other food intakes during treatment period is almost sure to cure diabetes. My BMI was less than 21 when I became diabetic. When I was cured of diabetes also my BMI was same. I have very strong hereditary factors in favor of diabetes. Both my parents were diabetic & continued to be so till death. My father died of hypoglycemia in the year 1958. Diabetes is a basic illness, similar to a voltage drop in an electronic or electrical circuit. That problem will adversely affect all related/connected sub-systems.
This does not mean that diabetes is incurable. GOOD LUCK ALL DIABETICS WORLD OVER.
Diabetes is CURABLE, but.[edit | edit source]
- Diabetes is indubitably curable.
- Cure is not as fast as Prof. Roy Taylor et al of Newcastle University UK claimed in their paper [1].
- The paper was presented at the American Diabetes Association conference on June 24, 2011 and published in Diabetologia (2011) 54:2506-2514.
- It has 46 References! (The article is also available online with open access (Distributed under the terms of the relative Commons Attribution Noncommercial License which permits any noncommercial use, distribution and reproduction in any medium, provided the original author(s) and source are credited) at Springerlink.com.)
- The fact is that the claim of cure of diabetes made in this paper [1] appears to be only partially true.
- It is vividly brought out in the second paragraph of second column of Diabetologia (2011) 54:2511.
That paragraph titled Post-intervention observation says: "At follow-up 12 weeks after completion of the dietary intervention...... HbA1c was unchanged (6.0 +/- 0.2 vs 6.2 +/- 0.1% [42 +/- 2 vs 44 +/- 1 mmol/mol]; p=0.10) and fasting plasma glucose increased modestly (5.7 +/- 0.5 vs 6.1 +/- 0.2 mmol/l; p<0.01), with a 2 h OGTT plasma glucose of 10.3 +/- 1.0 mmol/l. Three participants had recurrence of diabetes as judged by a 2 h post-load plasma glucose > 11.1 mmol/l......."
Elsewhere in the article [1] one finds values of "Fasting concentration" of Glucose for different cases and on different days during the course of this research. As these are expressed in "mmol/l" unit, an attempt is made to represent those in "mg/dl" which is more popular among laymen and hence easily comprehensible to common men. They are tabulated below.
Fasting Concentration | Controls | Baseline | Week 1 | Week 4 | Week 8 | Week 8+12 | 2Hr OGTT at Week 20 |
---|---|---|---|---|---|---|---|
Plasma glucose (mmol/l) | 5.3 +/- 0.1 | 9.2 +/- 0.4 | 5.9 +/- 0.4 | 5.7 +/- 0.6 | 5.7 +/- 0.5 | 6.1 +/- 0.2 | 10.3 +/- 1.0 |
Serum blood glucose (mg/dl) | 95.5 +/- 1.8 | 165.7 +/- 7.2 | 106.3 +/- 7.2 | 102.7 +/- 10.8 | 102.7 +/- 9.01 | 109.9 +/- 3.6 | 185.0 +/- 18.0 |
Comparison of these numbers with their specified limits given in the table below and taking note of the escalation of FBS in spite of "advice on portion size and healthy eating", clearly indicate that the cure is but a temporary phenomena in the case of this research, as explained in the "The pit-falls" section here. CURE is far far away, "long way to go before one can sleep".
Concentration | Fasting lower limit | Fasting upper limit | 2 hr PP | 2 hr OGTT |
---|---|---|---|---|
Plasma glucose (mmol/l) | 3.89 | 5.55 | 7.77 | 7.77 |
Serum blood glucose (mg/dl) | 70 | 100 | 140 | 140 |
The fact that 3 out of 7 claimed to be "cured" have already been reverted to diabetes in three months (2 h post-load plasma glucose > 11.1 mmol/l) makes the result statistically non-significant - though NOT insignificant (only 4 out of 11 = 36.36 %).
This does not mean or imply that diabetes be incurable. It only means that some more time and effort were required. The Newcastle University team did not complete the LAST MILE and hence missed the goal! Probably the ADA conference date persuaded them to prepare and make a declaration prematurely. Look at my results at (Lab results from July 2006.).
Results given above show that all the participants of this experiment are at the fag end of the last lap of their race towards recurrence of diabetes by the end of third month of "normal diet" with advice on portion size & healthy eating.
Further[edit | edit source]
Wikipedia in Interpretation of OGTT results says ... FPG should be below 6.1 mmol/l (110 mg/dl). Between 6.1 and 7.0 mmol/l (110 and 125 mg/dl) are "impaired fasting glycaemia", and repeatedly at or above 7.0 mmol/l (126 mg/dl) are diagnostic of diabetes... It continues: The 2 hour OGTT glucose level should be below 7.8 mmol/l (140 mg/dl). Levels between this and 11.1 mmol/l (200 mg/dl) indicate "impaired glucose tolerance". Glucose levels above 11.1 mmol/l (200 mg/dl) at 2 hours confirms a diagnosis of diabetes.
Measuring the UK study outcome with the W H O yardstick[edit | edit source]
The Wikipedia further says:
1999 WHO Diabetes criteria - Interpretation of OGTT Glucose levels
Glucose levels | NORMAL | NORMAL | IFGlycaemia | IFGlycaemia | IGTolerance | IGTolerance | DM | DM |
---|---|---|---|---|---|---|---|---|
Venous Plasma | Fasting | 2hrs | Fasting | 2hrs | Fasting | 2hrs | Fasting | 2hrs |
(mmol/l) | <6.1 | <7.8 | > 6.1 & <7.0 | <7.8 | <7.0 | >7.8 | >7.0 | >11.1 |
(mg/dl) | <110 | <140 | >110 & <126 | <140 | <126 | >140 | >126 | >200 |
I > Impaired, F > Fasting, G > Glucose, D > Diabetes, M > Mellitus
In the light of this WHO criteria, it becomes evident that no one of the eleven volunteers of the Newcastle study is in the NORMAL or Impaired Fasting Glycaemia levels. Out of the seven (7) claimed to have been cured, four (4) were reverted to Impaired Glucose Tolerance level (FPG < 7.0 mmol/l & 2hr OGTT > 7.8 mmol/l) and the rest three (3) were in the Confirmed Diagnosis of Diabetes Mellitus level (2hr OGTT > 11.1 mmol/l).
In brief
To put it in a nutshell, as per the 1999 WHO Diabetes criteria - Interpretation of OGTT Glucose levels no one of the eleven (11) volunteers of the Newcastle University study appears to be cured.
Followup by Dr. R. Taylor, NCU UK
http://web.archive.org/web/20171206124521/http://www.ncl.ac.uk:80/press/news/2015/10/type2diabetes/
https://www.sciencedaily.com/releases/2016/03/160322080542.htm
Visits[edit | edit source]
Till today May 31, 2015 number of visits is 83,001; Oct 17, 2015 it is 97,365; 6PM Nov 15, 2015 it is 100,111.
Contact[edit | edit source]
- Email: georgedappillygmailcom